细胞与分子免疫学杂志

参考文献

[1] Lin H,Ho A S,Haley-Vicente D,et al.Cloning and characterization of IL-1HY2,a novel interleukin-1 family member[J].J Biol Chem,2001,276(23):20597-20602.

[2] Wei Y,Xing J,Su X,et al.IL-38 attenuates myocardial ischemia-reperfusion injury by inhibiting macrophage inflammation[J].Immun Inflamm Dis,2023,11(6):e898.

[3] Ge Y,Chen J,Hu Y,et al.IL-38 alleviates inflammation in sepsis in mice by inhibiting macrophage apoptosis and activation of the NLRP3 inflammasome[J].Mediators Inflamm,2021,2021:6370911.

[4] Zhou X,Zhao R,Lv M,et al.ACSL4 promotes microglia-mediated neuroinflammation by regulating lipid metabolism and VGLL4 expression[J].Brain Behav Immun,2023,109:331-343.

[5] Han R T,Vainchtein I D,Schlachetzki J C M,et al.Microglial pattern recognition via IL-33 promotes synaptic refinement in developing corticothalamic circuits in mice[J].J Exp Med,2023,220(2):e20220605.

[6] 胡镝，谢雪梅，张徽.核受体相关转录因子1(Nurr1)对大鼠脑缺血再灌注损伤的神经保护作用及机制[J].细胞与分子免疫学杂志，2022,38(3):231-236.Hu S,Xie X,Zhang H.Protective effect of nuclear receptor related 1 (Nurr1) on nerves in rats with cerebral occlusion/reperfusion injury and its mechanism[J].Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi,2022,38(3):231-236.

[7] Garraud T,Harel M,Boutet M A,et al.The enigmatic role of IL-38 in inflammatory diseases[J].Cytokine Growth Factor Rev,2018,39:26-35.

[8] Tominaga M,Okamoto M,Kawayama T,et al.Overexpression of IL-38 protein in anticancer drug-induced lung injury and acute exacerbation of idiopathic pulmonary fibrosis[J].Respir Investig,2017,55(5):293-299.

[9] Dailing A,Mitchell K,Vuong N,et al.Characterization and validation of Arg286 residue of IL-1RAcP as a potential drug target for osteoarthritis[J].Front Chem,2020,8:601477.

[10] Chen Z,Giotti B,Kaluzova M,et al.A paracrine circuit of IL-1β/IL-1R1 between myeloid and tumor cells drives genotype-dependent glioblastoma progression[J].J Clin Invest,2023,133(22):e163802.

[11] Kim H,Hwang B,Cho S,et al.The ethanol extract of Cyperus exaltatus var.Iwasakii exhibits cell cycle dysregulation,ERK1/2/p38 MAPK/AKT phosphorylation,and reduced MMP-9-mediated metastatic capacity in prostate cancer models in vitro and in vivo[J].Phytomedicine,2023,114:154794.

[12] Jaiswal J,Egert J,Engesser R,et al.Mutual repression between JNK/AP-1 and JAK/STAT stratifies senescent and proliferative cell behaviors during tissue regeneration[J].PLoS Biol,2023,21(5):e3001665.

[13] Van De Veerdonk F L,Stoeckman A K,Wu G,et al.IL-38 binds to the IL-36 receptor and has biological effects on immune cells similar to IL-36 receptor antagonist[J].Proc Natl Acad Sci U S A,2012,109(8):3001-3005.

[14] Jiang L,Zhou X,Huang C,et al.The elevated expression of IL-38 serves as an anti-inflammatory factor in osteoarthritis and its protective effect in osteoarthritic chondrocytes[J].Int Immunopharmacol,2021,94:107489.

[15] Mora J,Schlemmer A,Wittig I,et al.Interleukin-38 is released from apoptotic cells to limit inflammatory macrophage responses[J].J Mol Cell Biol,2016,8(5):426-438.

[16] Dennis Graaf D M,Maas R J A,Smeekens S P,et al.Human recombinant interleukin-38 suppresses inflammation in mouse models of local and systemic disease[J].Cytokine,2021,137:155334.

[17] Boutet M A,Najm A,Bart G,et al.IL-38 overexpression induces anti-inflammatory effects in mice arthritis models and in human macrophages in vitro[J].Ann Rheum Dis,2017,76(7):1304-1312.

[18] Dinarello A,May M,Amo-Aparicio J,et al.IL-38 regulates intestinal stem cell homeostasis by inducing Wnt signaling and beneficial IL-1β secretion[J].Proc Natl Acad Sci U S A,2023,120(45):e2306476120.

[19] Hirota T,King B H.Autism spectrum disorder:A review[J].JAMA,2023,329(2):157-168.

[20] Safwi S R.Autism spectrum disorder in the US:Have we done enough?[J].Psychiatry Res,2023,330:115595.

[21] Maenner M J,Shaw K A,Bakian A V,et al.Prevalence and characteristics of autism spectrum disorder among children aged 8 years-autism and developmental disabilities monitoring Network,11 Sites,United States,2018[J].MMWR Surveill Summ,2021,70(11):1-16.

[22] Jiang C C,Lin L S,Long S,et al.Signalling pathways in autism spectrum disorder:mechanisms and therapeutic implications[J].Signal Transduct Target Ther,2022,7(1):229.

[23] Pan P Y,Taylor M J,Larsson H,et al.Genetic and environmental contributions to co-occurring physical health conditions in autism spectrum condition and attention-deficit/hyperactivity disorder[J].Mol Autism,2023,14(1):17.

[24] Ophir Y,Rosenberg H,Tikochinski R,et al.Screen time and autism spectrum disorder:A systematic review and meta-analysis[J].JAMA Netw Open,2023,6(12):e2346775.

[25] Roy M,Strate P.Autism spectrum disorders in adulthood-symptoms,diagnosis,and treatment[J].Dtsch Arztebl Int,2023,120(6):87-93.

[26] Gupta S,Ellis S E,Ashar F N,et al.Transcriptome analysis reveals dysregulation of innate immune response genes and neuronal activity-dependent genes in autism[J].Nat Commun,2014,5:5748.

[27] Patel A B,Tsilioni I,Leeman S E,et al.Neurotensin stimulates sortilin and mTOR in human microglia inhibitable by methoxyluteolin,a potential therapeutic target for autism[J].Proc Natl Acad Sci U S A,2016,113(45):E7049-E7058.

[28] Xiao S,Zhang Y,Liu Z,et al.Alpinetin inhibits neuroinflammation and neuronal apoptosis via targeting the JAK2/STAT3 signaling pathway in spinal cord injury[J].CNS Neurosci Ther,2023,29(4):1094-1108.

[29] Tsilioni I,Pantazopoulos H,Conti P,et al.IL-38 inhibits microglial inflammatory mediators and is decreased in amygdala of children with autism spectrum disorder[J].Proc Natl Acad Sci U S A,2020,117(28):16475-16480.

[30] Wang Q,Yang Q,Liu X.The microbiota-gut-brain axis and neurodevelopmental disorders[J].Protein Cell,2023,14(10):762-775.

[31] Chernikova M A,Flores G D,Kilroy E,et al.The brain-gut-microbiome system:Pathways and implications for autism spectrum disorder[J].Nutrients,2021,13(12):4497.

[32] Raghavan K,Dedeepiya V D,Yamamoto N,et al.Benefits of gut microbiota reconstitution by beta 1,3-1,6 glucans in subjects with autism spectrum disorder and other neurodegenerative diseases[J].J Alzheimers Dis,2023,94(s1):S241-S252.

[33] Andoh A,Nishida A.Pro- and anti-inflammatory roles of interleukin (IL)-33,IL-36,and IL-38 in inflammatory bowel disease[J].J Gastroenterol,2023,58(2):69-78.

[34] Saini V,Guada L,Yavagal D R.Global epidemiology of stroke and access to acute ischemic stroke interventions[J].Neurology,2021,97(20 Suppl 2):S6-S16.

[35] 杜佳琳，龚诗立，胡朝英，等.缺血性脑卒中的神经炎症反应机制及相关的抗炎药物研究进展[J].药物评价研究，2022,45(10):2126-2132.Du J,Gong S,Hu C,et al.Effect of pericytes on immune inflammatory response in ischemic stroke[J].Yao Wu Ping Jia Yan Jiu,2022,45(10):2126-2132.

[36] Gullotta G S,De Feo D,Friebel E,et al.Age-induced alterations of granulopoiesis generate atypical neutrophils that aggravate stroke pathology[J].Nat Immunol,2023,24(6):925-940.

[37] Fang J,Wang Z,Miao C Y.Angiogenesis after ischemic stroke[J].Acta Pharmacol Sin,2023,44(7):1305-1321.

[38] Benz A P,Hohnloser S H,Eikelboom J W,et al.Outcomes of patients with atrial fibrillation and ischemic stroke while on oral anticoagulation[J].Eur Heart J,2023,44(20):1807-1814.

[39] Romano J G,Rundek T.Expanding treatment for acute ischemic stroke beyond revascularization[J].N Engl J Med,2023,388(22):2095-2096.

[40] Ma?er B,Tsai A S,Einhaus J F,et al.Neuroimaging is the new “spatial omic”:multi-omic approaches to neuro-inflammation and immuno-thrombosis in acute ischemic stroke[J].Semin Immunopathol,2023,45(1):125-143.

[41] Cai W,Hu M,Li C,et al.FOXP3+ macrophage represses acute ischemic stroke-induced neural inflammation[J].Autophagy,2023,19(4):1144-1163.

[42] Zare Rafie M,Esmaeilzadeh A,Ghoreishi A,et al.IL-38 as an early predictor of the ischemic stroke prognosis[J].Cytokine,2021,146:155626.

[43] Zhang J,Zhao R,Chen J,et al.The effect of interleukin 38 on angiogenesis in a model of oxygen-induced retinopathy[J].Sci Rep,2017,7(1):2756.

[44] Liu X,Nemeth D P,Mckim D B,et al.Cell-type-specific interleukin 1 receptor 1 signaling in the brain regulates distinct neuroimmune activities[J].Immunity,2019,50(2):317-333.e6.

[45] Zhu H,Hu S,Li Y,et al.Interleukins and ischemic stroke[J].Front Immunol,2022,13:828447.

[46] Yang K,Bao T,Zeng J,et al.Research progress on pyroptosis-mediated immune-inflammatory response in ischemic stroke and the role of natural plant components as regulator of pyroptosis:A review[J].Biomed Pharmacother,2023,157:113999.

[47] Zhu J,Zhang J,Wang Y,et al.The effect of interleukin 38 on inflammation-induced corneal neovascularization[J].Curr Mol Med,2019,19(8):589-596.

[48] Gofton T E,Young G B.Sepsis-associated encephalopathy[J].Nat Rev Neurol,2012,8(10):557-566.

[49] Jiang J,Zou Y,Xie C,et al.Oxytocin alleviates cognitive and memory impairments by decreasing hippocampal microglial activation and synaptic defects via OXTR/ERK/STAT3 pathway in a mouse model of sepsis-associated encephalopathy[J].Brain Behav Immun,2023,114:195-213.

[50] Nasoni M G,Crinelli R,Iuliano L,et al.When nitrosative stress hits the endoplasmic reticulum:Possible implications in oxLDL/oxysterols-induced endothelial dysfunction[J].Free Radic Biol Med,2023,208:178-185.

[51] Wang J,Yang S,Jing G,et al.Inhibition of ferroptosis protects sepsis-associated encephalopathy[J].Cytokine,2023,161:156078.

[52] Huang X,Ye C,Zhao X,et al.TRIM45 aggravates microglia pyroptosis via Atg5/NLRP3 axis in septic encephalopathy[J].J Neuroinflammation,2023,20(1):284.

[53] Yue J,Tan Y,Huan R,et al.Mast cell activation mediates blood-brain barrier impairment and cognitive dysfunction in septic mice in a histamine-dependent pathway[J].Front Immunol,2023,14:1090288.

[54] Zhou S,Li Y,Hong Y,et al.Puerarin protects against sepsis-associated encephalopathy by inhibiting NLRP3/Caspase-1/GSDMD pyroptosis pathway and reducing blood-brain barrier damage[J].Eur J Pharmacol,2023,945:175616.

[55] Dehghan A,Dupuis J,Barbalic M,et al.Meta-analysis of genome-wide association studies in >80 000 subjects identifies multiple loci for C-reactive protein levels[J].Circulation,2011,123(7):731-738.

[56] Ge Y,Huang M,Wu Y,et al.Interleukin-38 protects against sepsis by augmenting immunosuppressive activity of CD4+CD25+ regulatory T cells[J].J Cell Mol Med,2020,24(2):2027-2039.

[57] Liu G,Guo J,Liu J,et al.Toll-like receptor signaling directly increases functional IL-17RA expression in neuroglial cells[J].Clin Immunol,2014,154(2):127-140.